Patients with sporadic FTLD exhibit similar increases in lysosomal proteins and storage material as patients with FTD due to GRN mutations (additional file 2)

Authors

Skylar E. Davis, University of Alabama at Birmingham
Anna K. Cook, University of Alabama at Birmingham
Justin A. Hall, University of Alabama at Birmingham
Yuliya Voskobiynyk, University of Alabama at Birmingham
Nancy V. Carullo, University of Alabama at Birmingham
Nicholas R. Boyle, University of Alabama at Birmingham
Ahmad R. Hakim, University of Alabama at Birmingham
Kristian M. Anderson, University of Alabama at Birmingham
Kierra P. Hobdy, University of Alabama at Birmingham
Derian A. Pugh, University of Alabama at Birmingham
Charles F. Murchison, University of Alabama at Birmingham
Laura J. McMeekin, Southern Research Institute
Micah Simmons, University of Alabama at Birmingham
Katherine A. Margolies, Southern Research Institute
Rita M. Cowell, University of Alabama at Birmingham
Alissa L. Nana, University of California, San Francisco
Salvatore Spina, University of California, San Francisco
Lea T. Grinberg, University of California, San Francisco
Bruce L. Miller, University of California, San Francisco
William W. Seeley, University of California, San Francisco
Andrew E. Arrant, University of Alabama at Birmingham

Author ORCID

Andrew E. Arrant 0000-0003-4706-9411

Publication Date

9-11-2024

Abstract

Additional file 2: Tables of patient data.

Keywords

Medicine, Cell Biology, Genetics, FOS: Biological sciences, Molecular Biology, Neuroscience, Immunology, FOS: Clinical medicine, Biological Sciences not elsewhere classified, Developmental Biology, Science Policy, Hematology

Related Datasets

Is supplement to: 10.1186/s40478-023-01571-4

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